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Pain and Neurogenic Inflammation (Progress in Inflammation Research) download ebook

by Philip K. Moore,Susan D. Brain

Pain and Neurogenic Inflammation (Progress in Inflammation Research) download ebook
ISBN:
0817658750
ISBN13:
978-0817658755
Author:
Philip K. Moore,Susan D. Brain
Publisher:
Birkhauser (March 1, 1999)
Language:
Pages:
344 pages
ePUB:
1257 kb
Fb2:
1539 kb
Other formats:
doc azw docx lrf
Category:
Medicine
Subcategory:
Rating:
4.6

Neurogenic inflammation is inflammation arising from the local release by afferent neurons of inflammatory mediators such as Substance P, Calcitonin Gene-Related Peptide (CGRP), neurokinin A (NKA), and endothelin-3 (ET-3)

Neurogenic inflammation is inflammation arising from the local release by afferent neurons of inflammatory mediators such as Substance P, Calcitonin Gene-Related Peptide (CGRP), neurokinin A (NKA), and endothelin-3 (ET-3). In such neurons, release of these pro-inflammatory mediators is thought to be triggered by the activation of ion channels that are the principal detectors of noxious enviornmental stimuli. In particular, the heat/capsaicin receptor TRPV1 and the irritant/wasabi receptor TRPA1.

Pain and Neurogenic Inflammation. Sensory neuropeptides, neurogenic inflammation and inflammatory cells. Joseph P. McGillis, Stefan Fernandez.

Over a hundred years ago it was recognised that primary sensory neurones play a crucially important "dual" role in inflammation

Pain and Neurogenic Inflammation book. Goodreads helps you keep track of books you want to read.

Pain and Neurogenic Inflammation book.

Explore publications in Neurogenic Inflammation, and find Neurogenic Inflammation experts. Inflammatory bowel diseases (IBD) have long been recognized to be accompanied by pain resulting in high morbidity. Transient receptor potential vanilloid 1 (TRPV1) and ankyrin 1 (TRPA1) ion channels located predominantly on the capsaicin-sensitive sensory neurons play a complex role in hyperalgesia and neurogenic inflammation.

Pain and Neurogenic Inflammation - ای بک کے مصنف .

Home Medical Books The Blood Brain Barrier and Inflammation . However, viruses, bacteria, parasites and auto-aggressive immune cells can penetrate the barrier and significantly contribute to CNS inflammation

However, viruses, bacteria, parasites and auto-aggressive immune cells can penetrate the barrier and significantly contribute to CNS inflammation. The BBB can actively contribute to neuroinflammation by presentation of chemokines, expression of cell adhesion molecules and alterations of barrier properties.